Difference between revisions of "Template:Bicalutamide metabolism"

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| link = Template:Bicalutamide metabolism
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| caption = {{{caption|}}}<noinclude>Example caption (this text isn't transcluded).</noinclude>
 
| caption = {{{caption|}}}<noinclude>Example caption (this text isn't transcluded).</noinclude>
 
| header = {{Navbar|Bicalutamide metabolism|mini=y}} {{No selflink|Bicalutamide}} metabolism in humans{{if pagename|Bicalutamide=<ref name="Cockshott2004" /><ref name="GrosseCampeau2013" />|other=<ref name="Bicalutamide metabolism review">{{cite journal | vauthors = Cockshott ID | title = Bicalutamide: clinical pharmacokinetics and metabolism | journal = Clinical Pharmacokinetics | volume = 43 | issue = 13 | pages = 855–878 | date = 2004 | pmid = 15509184 | doi = 10.2165/00003088-200443130-00003 | quote = These data indicate that direct glucuronidation is the main metabolic pathway for the rapidly cleared (''S'')-bicalutamide, whereas hydroxylation followed by glucuronidation is a major metabolic pathway for the slowly cleared (''R'')-bicalutamide.}}</ref><ref name="Bicalutamide glucuronidation via UGT1A9">{{cite journal | vauthors = Grosse L, Campeau AS, Caron S, Morin FA, Meunier K, Trottier J, Caron P, Verreault M, Barbier O | title = Enantiomer selective glucuronidation of the non-steroidal pure anti-androgen bicalutamide by human liver and kidney: role of the human UDP-glucuronosyltransferase (UGT)1A9 enzyme | journal = Basic & Clinical Pharmacology & Toxicology | volume = 113 | issue = 2 | pages = 92–102 | date  = August 2013 | pmid = 23527766 | pmc = 3815647 | doi = 10.1111/bcpt.12071 | quote = }}</ref>}}
 
| header = {{Navbar|Bicalutamide metabolism|mini=y}} {{No selflink|Bicalutamide}} metabolism in humans{{if pagename|Bicalutamide=<ref name="Cockshott2004" /><ref name="GrosseCampeau2013" />|other=<ref name="Bicalutamide metabolism review">{{cite journal | vauthors = Cockshott ID | title = Bicalutamide: clinical pharmacokinetics and metabolism | journal = Clinical Pharmacokinetics | volume = 43 | issue = 13 | pages = 855–878 | date = 2004 | pmid = 15509184 | doi = 10.2165/00003088-200443130-00003 | quote = These data indicate that direct glucuronidation is the main metabolic pathway for the rapidly cleared (''S'')-bicalutamide, whereas hydroxylation followed by glucuronidation is a major metabolic pathway for the slowly cleared (''R'')-bicalutamide.}}</ref><ref name="Bicalutamide glucuronidation via UGT1A9">{{cite journal | vauthors = Grosse L, Campeau AS, Caron S, Morin FA, Meunier K, Trottier J, Caron P, Verreault M, Barbier O | title = Enantiomer selective glucuronidation of the non-steroidal pure anti-androgen bicalutamide by human liver and kidney: role of the human UDP-glucuronosyltransferase (UGT)1A9 enzyme | journal = Basic & Clinical Pharmacology & Toxicology | volume = 113 | issue = 2 | pages = 92–102 | date  = August 2013 | pmid = 23527766 | pmc = 3815647 | doi = 10.1111/bcpt.12071 | quote = }}</ref>}}

Revision as of 06:45, 18 November 2018

Template:No selflink metabolism in humans
  1. REDIRECT Template:When pagename is
  • From a page move: This is a redirect from a page that has been moved (renamed). This page was kept as a redirect to avoid breaking links, both internal and external, that may have been made to the old page name.
(S)-Bicalutamide glucuronide
(R)-Hydroxybicalutamide
(R)-Hydroxybicalutamide glucuronide
via UGT1A9
via UGT1A9
via CYP3A4
Example caption (this text isn't transcluded).
50px Template documentation

To transclude this template, use:

{{Bicalutamide metabolism | caption = | align = }}
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